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central and peripheral nervous systems were intact. The severity of the degenerative changes was such that a developmental disorder of muscle could be excluded. Pearson and Fowler, in 1963, reported a brother and sister with similar clinical and pathologic ndings, and Walton et al described yet another patient, aged 4 years. By 1967 Vassella and colleagues were able to collect 27 cases from the medical literature and to add 8 cases of their own. The high incidence of sibling involvement pointed to an autosomal recessive inheritance. De ned as a muscle dystrophy already present at birth, often with contractures of proximal muscles and trunk, the severity of the weakness and degree of progression have varied widely. Of the 8 cases reported by Rotthauwe and others, one had a benign course, but the others all had weakness and hypotonia at birth, and dif culty in sucking and swallowing had interfered with nutrition. Their oldest patients, aged 14 and 23 years, and several others had walked, but at a late age. In the Finnish series of Donner and associates, congenital dystrophy accounted for 9 percent of the 160 cases of neuromuscular disease seen at their hospital over a decade. The weakness and hypotonia were generalized, and 3 had ECG abnormalities. The CK values were elevated and the EMGs were myopathic. This group of dystrophies began to come into focus in the 1960s with a series of articles from Japan relating the details in over 100 patients with congenital dystrophy (Fukuyama et al). It is the second most common muscular dystrophy in Japan but it is rare elsewhere. A feature of these cases was the coexistence of severe mental retardation and developmental anomalies of the cerebral cortex. Hyperlucency in the periventricular white matter (by

The carotid system consists of three major arteries the common carotid, internal carotid, and external carotid As indicated in Fig 34-2, the right common carotid artery arises at the level of the sternoclavicular notch from the innominate (brachiocephalic) artery, and the left common carotid comes directly from the aortic arch The common carotids ascend in the neck to the C4 level, just below the angle of the jaw, where each divides into external and internal branches (sometimes the bifurcation is slightly above or below this point) Common carotid occlusion accounts for less than 1 percent of cases of carotid artery syndrome the remainder being due to disease of the internal carotid artery itself Nevertheless, the common carotid can be occluded by an atheromatous plaque at its origin, more often on the left side.

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kitchens, and recreation facilities. The community center is the basic unit, administered by a commanding officer who directs the religious and social service activities. Officers receive college training, and the members, both men and women, are known as soldiers. One does not need to be a member, however, to work with the organization. A list from the Manhattan, New York, telephone book of Salvation Army branches gives you an idea of the organization s wideranging activities:

Atherosclerotic stenosis or occlusion of the midportion of the common carotid may also occur years after radiation therapy for laryngeal or other head and neck cancer If the bifurcation is patent, few if any symptoms may result, in some cases because retrograde ow from the external carotid maintains internal carotid ow and perfusion of the brain Because the syndromes caused by common carotid occlusion are identical to those of its internal branch, the remainder of this discussion is concerned with disease of the internal carotid artery The territory supplied by this vessel and its main branches is shown in Figs 34-4, 34-5, and 34-6 The clinical manifestations of atherosclerotic thrombotic disease of this artery are among the most variable of any cerebrovascular syndrome Unlike other cerebral vessels, the internal carotid artery is not an end vessel.

Figure 5.4 Kruskal s minimum spanning tree algorithm. procedure kruskal(G, w) Input: A connected undirected graph G = (V, E) with edge weights w e Output: A minimum spanning tree defined by the edges X for all u V : makeset(u) X = {} Sort the edges E by weight for all edges {u, v} E, in increasing order of weight: if find(u) = find(v): add edge {u, v} to X union(u, v)

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Fig. 14-6

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